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For a class of drugs already transforming millions of lives, this approach could further enhance both efficacy and tolerability, underscoring that responses to GLP-1 therapies are shaped not only by pharmacology but also by the subtle variations of the human genome

Tirzepatide, a dual GLP-1 and GIP receptor agonist, works through a different pathway than metformin's insulin-sensitizing effects, allowing both medications to coexist in a treatment plan when medically appropriate

Dysbiosis amplifies barrier dysfunction and immune activation, while inflammation further perturbs microbial composition and impairs epithelial repair, establishing a self-sustaining pathological cycle

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