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GLP-1RAs can reduce the development of macroalbuminuria and reduce the estimated glomerular filtration rate (eGFR) decline in participants with T2D [54, 55].This reduction is attributed to various mechanisms, including the reduction of oxidative stress and inflammation and inhibition of the immune systems response

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This triagonist approach combines GLP-1 and GIP receptor activity with glucagon-receptor signalling to modulate energy homeostasis and incretin pathways