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We conducted a wide range of additional control experiments to make sure we were looking at a true Orai1 deficiency phenotype. The investigators also created genetically modified mice that lacked Orai1 channels in excitatory neurons, but retained them elsewhere in the body and the brain

"Regenerative and Protective Actions of the GHK-Cu Peptide in the Light of the New Gene Data." International Journal of Molecular Sciences , 19(7), 1987

Tas var{2 regult asinsvadu endotlija auganas faktora (VEGF) un epidermas auganas faktora (EGF) izpausmes un uzlabot gotdas barjeras funkciju