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High-dose ferric ammonium citrate (FeAC) triggers MM cell death with concurrent elevation of MDA - a lipid peroxidation byproduct and hallmark of ferroptosis, establishing iron metabolism as a critical modulator in MM pathophysiology (Nemeth and Ganz, 2023), MM patients exhibit paradoxical hepcidin downregulation or functional impairment during iron overload, creating a self-reinforcing cycle of iron accumulation that exacerbates disease progression (Maes et al., 2010