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Current notable studies focus on its potential in treating infected or injured eyes, such as cases involving Pseudomonas aeruginosa -induced keratitis, and the facilitation of corneal wound healing [5, 6]

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This inextricably links gliotoxin/BmGT formation to the cellular Methyl/Met cycle via GtmA (Owens et al., 2015) (Figure 2) and leads to the conclusion that biosynthesis and dissipation of the disulfide-containing non-ribosomal peptide gliotoxin requires significant utilization of cellular sulfur-containing metabolites, GSH and SAM (Figure 2)