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For example, ROS, via oxidation of thioredoxin, enhance the nuclear localization of both the ataxia-telangiectasia mutated (ATM) serine/threonine kinase, which is involved in DNA damage repair 31,32 , and redox factor-1 (Ref-1), a multifunctional protein that enables Fos/Jun DNA-binding and is identical to the apurinic/apyrimidinic 1 (APE1) endonuclease 33

(11) demonstrated that interferon-gamma (IFN-) could upregulate CUL5 expression and disrupt the binding of Cdc37/Hsp90 to HER2, consequently inducing tumor cell senescence and promoting HER2 degradation

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