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There is a theoretical concern that aggressive senolytic clearance could remove this anti-tumor senescence signal, potentially accelerating tumor growth in individuals with pre-malignant cells

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Mitochondrial transfer mediated by mesenchymal stem cells (MSCs) could restore the mitochondrial pool of neurons and inhibit ferroptosis, 951 providing a promising clinical translational strategy for ferroptosis-related central nervous system diseases based on stem cell-mediated mitochondrial therapy