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In normal condition, the antioxidant defense system should minimize the levels of reactive oxygen species (ROS) produced during diabetes mellitus while allowing ROS useful roles of cell signaling and redox regulation [2]
Biol Bull Rev 13(2):8191
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Moreover, Cyp2e1-/- mice showed resistance to the APAP high dose-associated hepatotoxicity, whereas human CYP2E1 transgenic mice were susceptible, suggesting that CYP2E1 is the major P450 enzyme participating in APAP activation (Cheung et al., 2005)
