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Most gastrointestinal effects ease as the body adjusts over the first weeks

(1) This may upregulate growth hormone (GH) synthesis, which is a hormone with catabolic potential towards fat cells, and also may possess anabolic potential via a mediator called insulin-like growth factor 1 (IGF-1) The research suggests that it may produce rapid, concentration-dependent, pulsatile GH elevations, yielding GH pulses roughly 20- to 30-fold above basal values and potentially close to other GHSs such as GHRP-6 and GHRP-2

Their mechanism of action involves several facets: initially, they inhibit the production and release of inflammatory mediators such as prostaglandins, leukotrienes, and tumor necrosis factor, which are pivotal in the pathogenesis of DED (Sheppard et al., 2013)

For scientists and clinicians, its an excellent candidate for further study